Publication: A Case of Primary Hypoparathyroidism Presenting with Acute Kidney Injury Secondary to Rhabdomyolysis.
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Authors
SUMNU, A
AYDIN, Z
GURSU, MELTEM
UZUN, S
KARADAG, S
CEBECI, E
OZTURK, S
Kazancioglu, RÜMEYZA
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Abstract
Symmetric calcification of the basal ganglia identified radiographically occurs in a variety of familial and nonfamilial
conditions. Primary Familial Brain Calcifications (PFBC),
which were known by many names previously, including Fahr disease and striopallidodentate calcinosis, are a
genetic disease characterized by various mutations in four
separate genes and autosomal dominant inheritance [1–4].
PFBC may present with various psychiatric and neurological
symptoms [5]. On the other hand, many secondary causes,
either infectious, toxic, or metabolic, have been described
to cause symmetrical basal ganglion calcifications and so
are in the differential diagnosis of PFBC [6]. Parathyroid
diseases such as hypoparathyroidism, pseudohypoparathyroidism, and pseudo-pseudohypoparathyroidism are in the
forefront among the metabolic causes. Idiopathic or postsurgical hypoparathyroidism is the most common cause of
symmetric calcification of the basal ganglia [7–9]. Herein,
a case of primary hypoparathyroidism with severe tetany,
rhabdomyolysis, and acute kidney injury (AKI) is presented.
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SUMNU A., AYDIN Z., GURSU M., UZUN S., KARADAG S., CEBECI E., OZTURK S., Kazancioglu R., -A Case of Primary Hypoparathyroidism Presenting with Acute Kidney Injury Secondary to Rhabdomyolysis.-, Case reports in nephrology, cilt.2016, ss.3240131, 2016