Publication:
May TLR4 Asp299Gly and IL17 His161Arg polymorphism be associated with progression of primary measles infection to subacute sclerosing panencephalitis?

dc.contributor.authorKarakas-Celik, Sevim
dc.contributor.authorPiskin, Ibrahim Etem
dc.contributor.authorKeni, Mehmet Fatih
dc.contributor.authorCalik, Mustafa
dc.contributor.authorIscan, AKIN
dc.contributor.authorDursun, Ahmet
dc.contributor.institutionauthorİŞCAN, AKIN
dc.date.accessioned2019-10-05T13:13:07Z
dc.date.available2019-10-05T13:13:07Z
dc.date.issued2014-09-01
dc.description.abstractSSPE is a progressive neurological disorder of children. Only some of the children who are infected with measles virus develop SSPE, which supports individual variation. TLR-2 and TLR-4 play an important role in innate immunity by recognizing envelope proteins of MV. Another important cytokine that plays an important role in orchestrating innate immune function is IL-17. The purpose of our study is to elucidate whether the TLR2, TLR4, IL17F and IL17A gene polymorphisms are susceptibility genes for the development of SSPE.
dc.identifier10.1016/j.ejrad.2004.05.005
dc.identifier.citationKarakas-Celik S., Piskin I. E. , Keni M. F. , Calik M., Iscan A., Dursun A., -May TLR4 Asp299Gly and IL17 His161Arg polymorphism be associated with progression of primary measles infection to subacute sclerosing panencephalitis?-, GENE, cilt.547, ss.186-190, 2014
dc.identifier.doi10.1016/j.gene.2014.03.056
dc.identifier.pubmed24690400
dc.identifier.scopus84904437362
dc.identifier.urihttps://hdl.handle.net/20.500.12645/1632
dc.identifier.wosWOS:000340327800002
dc.language.isoen
dc.titleMay TLR4 Asp299Gly and IL17 His161Arg polymorphism be associated with progression of primary measles infection to subacute sclerosing panencephalitis?
dc.typeArticle
dspace.entity.typePublication
local.article.journalnameEUROPEAN JOURNAL OF RADIOLOGY
local.avesis.id272c831c-86e7-4c1a-aa16-6d192f9098f8
local.avesis.response1502
local.publication.goal03 - Sağlık ve Kaliteli Yaşam
local.publication.isinternational1
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relation.isAuthorOfPublication.latestForDiscovery12538a20-d5a6-4246-ab37-58afe7bbae72
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relation.isGoalOfPublication.latestForDiscovery9c198c48-b603-4e2f-8366-04edcfc1224c
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