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The predictive value of M30 and oxidative stress for left ventricular remodeling in patients with anterior ST-segment elevation myocardial infarction treated with primary percutaneous coronary intervention

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dc.contributor.authorTURKOGLU, Caner
dc.contributor.authorGUR, Mustafa
dc.contributor.authorSEKER, Taner
dc.contributor.authorSelek, Sahbettin
dc.contributor.authorKocyigit, ABDÜRRAHİM
dc.contributor.institutionauthorSELEK, ŞAHABETTİN
dc.contributor.institutionauthorKOÇYİĞİT, ABDÜRRAHİM
dc.date.accessioned2019-10-05T15:23:46Z
dc.date.available2019-10-05T15:23:46Z
dc.date.issued2016-12-01
dc.description.abstractBackground Left ventricular (LV) remodeling is an important pathophysiological event that develops following acute myocardial infarction and causes LV systolic dysfunction. Mechanisms such as apoptosis, necrosis, and oxidative stress play an important role in LV remodeling.
dc.identifier10.1007/s11605-009-1120-3
dc.identifier.citationTURKOGLU C., GUR M., SEKER T., Selek S., Kocyigit A., -The predictive value of M30 and oxidative stress for left ventricular remodeling in patients with anterior ST-segment elevation myocardial infarction treated with primary percutaneous coronary intervention-, CORONARY ARTERY DISEASE, cilt.27, ss.690-695, 2016
dc.identifier.doi10.1097/mca.0000000000000416
dc.identifier.pubmed27479531
dc.identifier.scopus84980390600
dc.identifier.urihttps://hdl.handle.net/20.500.12645/6195
dc.identifier.wosWOS:000387489000010
dc.language.isoen
dc.titleThe predictive value of M30 and oxidative stress for left ventricular remodeling in patients with anterior ST-segment elevation myocardial infarction treated with primary percutaneous coronary intervention
dc.typeArticle
dspace.entity.typePublication
local.article.journalnameJOURNAL OF GASTROINTESTINAL SURGERY
local.avesis.id99e9e4f5-6926-4e00-8da1-fff111e644a7
local.avesis.response6065
local.publication.isinternational1
relation.isAuthorOfPublication8eb7d539-8ea4-435c-830e-8ef630342c7d
relation.isAuthorOfPublication75babbd2-f46b-4121-aa00-54d883a6f907
relation.isAuthorOfPublication.latestForDiscovery8eb7d539-8ea4-435c-830e-8ef630342c7d
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