Person: DENİZ, ÇİĞDEM
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Publication Metadata only Direct Catheter-Based Thrombectomy for Acute Ischemic Stroke Outcomes of Consecutive Patients Treated in Interventional Cardiology Centers in Close Cooperation With Neurologists(2015-07-28) WIDIMSKY, Petr; Asil, TALİP; ABELSON, Mark; KOZNAR, Boris; TASAL, Abdurrahman; ROOS, Johan; VASKO, Peter; PEISKER, Tomas; Deniz, Cigdem; VAVROVA, Jana; YAMAC, Hatice Aylin; STETKAROVA, Ivana; AY, Nuray Kahraman; Bacaksiz, AHMET; TUZGEN, Saffet; MALY, Marek; GOKTEKIN, Omer; ASİL, TALIP; DENİZ, ÇİĞDEM; BACAKSIZ, AHMET; KAHRAMAN AY, NURAYPublication Metadata only PROLONGED HYPOTENSION AFTER CAROTID ARTERY STENTING: INCIDENCE, PREDICTORS AND CONSEQUENCES(2016-10-01) GOKCAL, ELİF; NIFTALIYEV, E.; DENIZ, C.; ERGELEN, M.; GUZEL, VİLDAN; GOKTEKIN, O.; Asil, TALİP; GÖKÇAL, ELİF; DENİZ, ÇİĞDEM; GÜZEL, VİLDAN; ASİL, TALIPPublication Metadata only Epilepsy in Elderly Patients(2015-01-01T00:00:00Z) Halac, Gulistan; BİLGEN, HALİDE RENGİN; Kocaman, Gulsen; Niftaliyev, Elvin; DENİZ, ÇİĞDEM; ASİL, Talip; BİLGEN, HALİDE RENGİN; DENİZ, ÇİĞDEM; ASİL, TALIPObjectives: The incidence of new-onset epilepsy is high among the elderly. This study presents the demographic characteristics, etiological factors, seizure types and response rates to treatment in a group of patients aged 65 years or over with delayed-onset epilepsy.Publication Metadata only Association between cerebrovascular reactivity and severity of diabetic retinopathy in patients with diabetes mellitus(2015-04-01) YAMAN, A.; GULTEKIN, T. Ozdemir; DURUYEN, H.; ALTINISIK, M.; DENIZ, C.; Asil, TALİP; DENİZ, ÇİĞDEM; ASİL, TALIPPublication Metadata only HYPERCOAGULOPATHYON PATIENT WITH CAROTID AND VERTEBRAL ARTERY DISSECTION(2016-10-01) DENIZ, C.; BILGEN, H. R.; HALAC, G.; GULTEKIN, T. Ozdemir; GOKTEKIN, O.; Asil, TALİP; DENİZ, ÇİĞDEM; ASİL, TALIPPublication Metadata only Progressive deficit in isolated pontine infarction: the association with etiological subtype, lesion topography and outcome(2017-09-01T00:00:00Z) GÖKÇAL, ELİF; Niftaliyev, Elvin; Baran, Gozde; DENİZ, ÇİĞDEM; ASİL, Talip; GÖKÇAL, ELİF; BARAN, GÖZDE; DENİZ, ÇİĞDEM; ASİL, TALIPIt is important to predict progressive deficit (PD) in isolated pontine infarction, a relatively common problem of clinical stroke practice. Traditionally, lacunar infarctions are known with their progressive course. However, few studies have analyzed the branch atheromatous disease subtype as a subtype of lacunar infarction, separately. There are also conflicting results regarding the relationship with the topography of lesion and PD. In this study, we classified etiological subtypes and lesion topography in isolated pontine infarction and aimed to investigate the association of etiological subtypes, lesion topography and clinical outcome with PD. We analyzed demographics, laboratory parameters, and risk factors of 120 patients having isolated pontine infarction and admitted within 24 h retrospectively. PD was defined as an increase in the National Institutes of Health Stroke scale >= 2 units in 5 days after onset. Patients were classified as following: large artery disease (LAA), basilar artery branch disease (BABD) and small vessel disease (SVD). Upper, middle and lower pontine infarcts were identified longitudinally. Functional outcome at 3 months was determined according to modified Rankin scores. Of 120 patients, 41.7% of the patients were classified as BABD, 30.8% as SVD and 27.5% as LAA. 23 patients (19.2%) exhibited PD. PD was significantly more frequent in patient with BABD (p 0.006). PD was numerically higher in patients with lower pontine infarction. PD was associated with BABD and poor functional outcome. It is important to discriminate the BABD neuroradiologically from other stroke subtypes to predict PD which is associated with poor functional outcome in patients with isolated pontine infarctions.