SUMNU, AAYDIN, ZGURSU, MELTEMUZUN, SKARADAG, SCEBECI, EOZTURK, SKazancioglu, RÜMEYZA2019-10-052019-10-052016-01-01SUMNU A., AYDIN Z., GURSU M., UZUN S., KARADAG S., CEBECI E., OZTURK S., Kazancioglu R., -A Case of Primary Hypoparathyroidism Presenting with Acute Kidney Injury Secondary to Rhabdomyolysis.-, Case reports in nephrology, cilt.2016, ss.3240131, 2016https://hdl.handle.net/20.500.12645/4961Symmetric calcification of the basal ganglia identified radiographically occurs in a variety of familial and nonfamilial conditions. Primary Familial Brain Calcifications (PFBC), which were known by many names previously, including Fahr disease and striopallidodentate calcinosis, are a genetic disease characterized by various mutations in four separate genes and autosomal dominant inheritance [1–4]. PFBC may present with various psychiatric and neurological symptoms [5]. On the other hand, many secondary causes, either infectious, toxic, or metabolic, have been described to cause symmetrical basal ganglion calcifications and so are in the differential diagnosis of PFBC [6]. Parathyroid diseases such as hypoparathyroidism, pseudohypoparathyroidism, and pseudo-pseudohypoparathyroidism are in the forefront among the metabolic causes. Idiopathic or postsurgical hypoparathyroidism is the most common cause of symmetric calcification of the basal ganglia [7–9]. Herein, a case of primary hypoparathyroidism with severe tetany, rhabdomyolysis, and acute kidney injury (AKI) is presented.eninfo:eu-repo/semantics/openAccessArticle10.1155/2016/324013127034860trdizin