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TAŞLIDERE, ELİF

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Now showing 1 - 9 of 9
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    Therapeutic effects of melatonin and quercetin on carbon tetrachloride-induced cardiac damage in rats
    (2016-03-01) ELBE, HÜLYA; EŞREFOĞLU, MUKADDES; CETIN, A; TAŞLIDERE, ELİF; ATES, BURHAN; EŞREFOĞLU, MUKADDES; TAŞLIDERE, ELİF
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    Protective effect of melatonin and quercetin in carbon tetrachloride induced lung injury
    (2014-01-21) TAŞLIDERE, ELİF; ELBE, HÜLYA; ÇETİN, ASLI; EŞREFOĞLU, MUKADDES; TAŞLIDERE, ELİF; EŞREFOĞLU, MUKADDES
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    Retikulum hücreleri
    (2012-05-01) TAŞLIDERE, ELİF; EŞREFOĞLU, MUKADDES; TAŞLIDERE, ELİF; EŞREFOĞLU, MUKADDES
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    Melatonin is effective in reducing stress-induced organ damage in Wistar albino rats
    (2014-01-01T00:00:00Z) Esrefoglu, MUKADDES; AKINCI, Aysin; Elbe, Hulya; TAŞLIDERE, ELİF; Cetin, Asli; ATEŞ, BURHAN; EŞREFOĞLU, MUKADDES; TAŞLIDERE, ELİF
    In the present study, we tried to investigate the effects of melatonin, a novel antioxidant and a potent free radical scavenger, in stress-induced cerebral, cerebellar, cardiac, and hepatic oxidative damage using microscopic and biochemical analysis. A total of 32 male Wistar albino rats were divided into control, stress, stress + saline, and stress + melatonin groups. The rats from the stress groups were exposed to high stress conditions of starvation, immobilization, and cold exposure. The rats from the stress + melatonin group received melatonin daily at 20 mg/kg body weight intraperitoneally for 7 days. At the end of the experiment, the brain, cerebellum, heart, and liver were rapidly removed. The main histopathological damage scores (MHDSs) of the stress and stress + saline groups were higher than those of control group for all of the organs. The MHDSs of melatonin-administered group were lower than those of stress and stress + saline groups. The main tissue superoxide dismutase activities of the stress + melatonin group were even higher than those of the control group in the cerebellum and liver, and main tissue catalase activities of the stress + melatonin group were even higher than those of control group in all of the organs. As a conclusion, we found melatonin very effective in reducing stress-induced organ damage by inhibiting lipid peroxidation and supporting the cellular antioxidant defense system.
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    Preventive Effects of Resveratrol against Azoxymethane Induced Testis Injury in Rats
    (2016-06-20) KURUŞ, MELTEM; BAY KARABULUT, AYSUN; TAŞLIDERE, ELİF; OTLU, ÖNDER; TAŞLIDERE, ELİF
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    The Effect of Ceftriaxone on Testicular Connexin 43 Expression.
    (2016-03-01T00:00:00Z) Sahin, Levent; Sahin, Hilal; VARDI, NİGAR; Karahan, Feride; YILDIZ, AZİBE; TAŞLIDERE, ELİF; GÜL, SEMİR; TAŞLIDERE, ELİF
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    Development of Liver and Pancreas
    (2017-01-01) EŞREFOĞLU, MUKADDES; TAŞLIDERE, ELİF; CETIN, ASLI; EŞREFOĞLU, MUKADDES; TAŞLIDERE, ELİF
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    Protective effect of melatonin and quercetin on hepatic damage induced by carbon tetrachloride in wistar albino rats.
    (2012-06-29) ÇETİN, ASLI; TAŞLIDERE, ELİF; ELBE, HÜLYA; EŞREFOĞLU, MUKADDES; TAŞLIDERE, ELİF; EŞREFOĞLU, MUKADDES
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    Beta-glucan attenuates cerebral ischemia/reperfusion-induced neuronal injury in a C57BL/J6 mouse model
    (2019-01-01T00:00:00Z) KAYA, KÜRŞAT; ÇİFTÇİ, OSMAN; ÖZTANIR, MUSTAFA NAMIK; TAŞLIDERE, ELİF; BAŞAK TÜRKMEN, NEŞE; TAŞLIDERE, ELİF
    Beta-glucans (beta g), that have many useful effects on human health, are natural polysaccharides. Our aim in this study was to determine useful effect of beta g against oxidative and neuronal damage caused by global cerebral ischemia/reperfusion (IR) in stroke imitated mice via surgical operation. A total of 40 mice divided into four equal groups randomly. The group 1 (sham operated) was kept as control. Bilateral carotid arteries of subjects in group 2 (I/R) and group 4 (I/R + beta g) were clipped for 15 min, and the mice in group 4 (I/R + beta g) were treated with beta g (50 mg/kg/day), while the mice in group 2 (I/R) were treated with only vehicle for 10 days. The mice of group 3 (beta g) were treated with beta g for 10 days without carotid occlusion. Global cerebral I/R significantly increased oxidative stress and decreased members of anti-oxidant defense system. In addition, I/R caused histopathological damage in the brain tissue. However, beta g treatment ameliorated both oxidative and histopathological effects of I/R. Our present study showed that beta g treatment significantly ameliorated oxidative and histological damage in the brain tissue caused by cerebral I/R. Therefore, beta g treatment can be used as supportive care for ischemic stroke patients.